Galactose mutarotase

GALM
Available structures
PDB	Ortholog search: PDBe RCSB
List of PDB id codes
	1SNZ, 1SO0
Identifiers
Aliases	GALM, BLOCK25, GLAT, HEL-S-63p, IBD1, galactose mutarotase (aldose 1-epimerase), galactose mutarotase
External IDs	OMIM: 137030 MGI: 2442420 HomoloGene: 71795 GeneCards: GALM
Gene location (Human)
Chr.	Chromosome 2 (human)[1]
End	38,741,237 bp[1]
Gene location (Mouse)
Chr.	Chromosome 17 (mouse)[2]
End	80,185,101 bp[2]
Gene ontology
Molecular function	• isomerase activity; • aldose 1-epimerase activity; • carbohydrate binding; • catalytic activity;
Cellular component	• extracellular exosome; • cytoplasm;
Biological process	• hexose metabolic process; • glucose metabolic process; • galactose metabolic process; • galactose catabolic process via UDP-galactose; • carbohydrate metabolic process;
	Sources:Amigo / QuickGO
Orthologs
	130589
	319625
	ENSG00000143891
	ENSMUSG00000035473
	Q96C23
	Q8K157
	NM_138801
	NM_176963
	NP_620156
	NP_795937
	Wikidata
View/Edit Human	View/Edit Mouse

Galactose mutarotase (aldose 1-epimerase) (gene name GALM) is a human enzyme that converts alpha-aldose to the beta-anomer. It belongs to family of aldose epimerases.

External links

PDBe-KB provides an overview of all the structure information available in the PDB for Human Aldose 1-epimerase (Galactose mutarotase)

GRCh38: Ensembl release 89: ENSG00000143891 - Ensembl, May 2017
GRCm38: Ensembl release 89: ENSMUSG00000035473 - Ensembl, May 2017
"Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
"Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.

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[refGRCh38Ensembl-1] GRCh38: Ensembl release 89: ENSG00000143891 - Ensembl, May 2017

[refGRCm38Ensembl-2] GRCm38: Ensembl release 89: ENSMUSG00000035473 - Ensembl, May 2017

[3] "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.

[4] "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.